Table 2 Illustration example for the system workflow of protein coreference resolution
From: Improving protein coreference resolution by simple semantic classification
Example text (PMID-7964516) | T cell hybridomas respond to activation signals by undergoing apoptotic cell death, and this is likely to represent comparable events related to tolerance induction in immature and mature T cells in vivo. Previous studies using antisense oligonucleotides implicated the c-Myc protein in the phenomenon of activation-induced apoptosis. This role for c-Myc in apoptosis is now confirmed in studies using a dominant negative form of its heterodimeric binding partner, Max, which we show here inhibits activation-induced apoptosis. |
Preprocessing results: sentences and chunks (partially) (Step 0) | S1: T cell hybridomas respond to activation signals by undergoing apoptotic cell death, and this is likely to represent comparable events related to tolerance induction in immature and mature T cells in vivo. S2: Previous studies using antisense oligonucleotides implicated the c-Myc protein in the phenomenon of activation-induced apoptosis. S3: [This [[role] for [c-Myc] in [apoptosis]]] is now confirmed in [[studies] using a dominant negative form of its heterodimeric binding partner, Max, which we show here inhibits activation-induced apoptosis]. |
Markables (Step 1) | S1: [T cell hybridomas] respond to [activation signals] by undergoing [apoptotic cell death], and this is likely to represent [comparable events related to tolerance induction] in [immature and mature T cells [in vivo]]. S2: [Previous studies using [antisense oligonucleotides]] implicated [the c-Myc protein] in [the phenomenon of [activation-induced apoptosis]]. S3: [This role for [c-Myc] in [apoptosis]] is now confirmed in [studies] using a [dominant negative form of [[its] heterodimeric binding partner,[Max]]], [which] [we] show here inhibits [activation-induced apoptosis]. |
Anaphors (Step 2) | S1: T cell hybridomas respond to activation signals by undergoing apoptotic cell death, and [this] is likely to represent comparable events related to tolerance induction in immature and mature T cells in vivo. S2: Previous studies using antisense oligonucleotides implicated the c-Myc protein in the phenomenon of activation-induced apoptosis. S3: This role for c-Myc in apoptosis is now confirmed in studies using a dominant negative form of [its] heterodimeric binding partner, Max, [which] we show here inhibits activation-induced apoptosis. |
Antecedent candidates of its (Step 3) | S1: [T cell hybridomas] respond to [activation signals] by undergoing [apoptotic cell death], and this is likely to represent [comparable events related to tolerance induction] in [immature and mature T cells [in vivo]]. S2: [Previous studies using [antisense oligonucleotides]] implicated [the c-Myc protein] in [the phenomenon of [activation-induced apoptosis]]. S3: [This role for [c-Myc] in [apoptosis]] is now confirmed in [studies] using [a dominant negative form of its heterodimeric binding partner, Max], which we show here inhibits activation-induced apoptosis. |
Predicted antecedent of its (Step 4) | S1: T cell hybridomas respond to activation signals by undergoing apoptotic cell death, and this is likely to represent comparable events related to tolerance induction in immature and mature T cells in vivo. S2: Previous studies using antisense oligonucleotides implicated the c-Myc protein in the phenomenon of activation-induced apoptosis. S3: This role for [c-Myc] in apoptosis is now confirmed in studies using a dominant negative form of its heterodimeric binding partner, Max, which we show here inhibits activation-induced apoptosis. |